By Collin, Catherine
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Postulates regarding pathogenesis, such as oxidative stress and excitotoxicity, have led to the discovery of abnormal mitochondrial function in PD and a search for biochemical markers. Functional imaging studies have detected subclinical nigral dopaminergic dysfunction in individuals at risk of developing PD. Current symptomatic therapies are aimed at enhancing dopaminergic transmission. However, some commonly used PD medications may alternative reactions with both symptomatic and neuroprotec*To whom correspondence should be addressed.
